Metabolic alterations connected to T2D are very well characterised by epidemiological and research centered experiments. The pathogenesis and development of T2D is ascribed to four mechanisms; greater advanced glycation conclude products (AGE) development, amplified polyol pathway flux, activation of protein kinase C (PKC) isoforms, and greater hexosamine pathway flux[seven]. Until https://www.directivepublications.org/journal-of-diabetology-research/